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Journal of Dental Research
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Responses of the Human Submandibular Artery to ACh and VIP

D. Stojic1,*, S. Pesic2, M. Radenkovic3, J. Popovic-Roganovic1, Z. Pesic4 and L. Grbovic3

1 Department of Pharmacology, Faculty of Stomatology, University of Belgrade, Dr. Subotica br. 8, 11 000 Belgrade;
2 Department of Pharmacology, Medical Faculty, University of Nis;
3 Department of Pharmacology, Clinical Pharmacology and Toxicology, Medical Faculty, University of Belgrade; and
4 Department of Maxillofacial Surgery, Medical Faculty, University of Nis, Serbia and Montenegro


Figure 1
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  		Figure 1. The effects of endothelial denudation (A) and L-NOARG, 10–5 mol/L (B), on ACh-produced relaxant action in the human submandibular artery. Each point represents mean ± SEM (n = 4). Responses are expressed as percentages of the phenylephrine pre-contraction.

 

Figure 2
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  		Figure 2. The effects of indomethacin, 10–5 mol/L (A), concomitant incubation of indomethacin, 10–5 mol/L, and L-NOARG, 10–5 mol/L (B), and 4-aminopyridine, 10–5 mol/L (C), on ACh-produced relaxant action in the human submandibular artery. Each point represents mean ± SEM (n = 4). Responses are expressed as percentages of the phenylephrine pre-contraction.

 

Figure 3
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  		Figure 3. The effects of endothelial denudation (A) and L-NOARG, 10–5 mol/L (B), on VIP-produced relaxant action in the human submandibular artery. Each point represents mean ± SEM (A, n = 7; B, n = 5). Responses are expressed as percentages of the phenylephrine pre-contraction.

 

Figure 4
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  		Figure 4. The effects of indomethacin, 10–5 mol/L (A), 4-aminopyridine, 10–5 mol/L (B), and forskolin, 10–5 mol/L (C), on VIP-produced relaxant action in the human submandibular artery. Each point represents mean ± SEM (A and C, n = 4; B, n = 5). Responses are expressed as percentages of the phenylephrine pre-contraction.

 

Journal of Dental Research, Vol. 86, No. 6, 565-570 (2007)
DOI: 10.1177/154405910708600615
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