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Journal of Dental Research
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Guiding Periodontal Pocket Recolonization: a Proof of Concept

W. Teughels1,*, M.G. Newman2, W. Coucke1, A.D. Haffajee3, H.C. Van Der Mei4, S. Kinder Haake2, E. Schepers5, J.-J. Cassiman6, J. Van Eldere7, D. van Steenberghe1 and M. Quirynen1

1 Catholic University Leuven, Research Group for Microbial Adhesion, Department of Periodontology, Kapucijnenvoer 7, 3000 Leuven, Belgium;
2 UCLA, School of Dentistry, 10833 Le Conte Avenue, Los Angeles, CA, USA;
3 The Forsyth Institute, Department of Periodontics, 140 The Fenway, Boston, MA, USA;
4 University of Groningen, Department of Biomedical Engineering, Antonius Deusinglaan 1, 9713 AV Groningen, the Netherlands;
5 Catholic University Leuven, BioMat Research Cluster, Kapucijnenvoer 7, 3000 Leuven, Belgium;
6 Catholic University Leuven, Department of Human Genetics, Herestraat 49, 3000 Leuven, Belgium; and
7 Catholic University Leuven, Centre for Molecular Diagnostics, Herestraat 49, 3000 Leuven, Belgium


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  		Figure. Mean change (± standard error) in subgingival bacterial load (n = 16/treatment group and time-point) from baseline (BL), and 2, 4, 8, and 12 wks later in each treatment group [Nc = negative control (no treatment), Rp = subgingival debridement via root planing, Rpsingle = root planing + single application of a mixture of beneficial bacteria, Rpmulti = root planing + multiple applications of a mixture of beneficial bacteria]. * represents a treatment strategy that is significantly different from Nc (p < 0.05). Arrows represent significant differences among treatment strategies (p < 0.05). (a) Total number of anaerobic species. (b) Total number of black-pigmented bacteria. (c) Total number of aerobic species. (d) Total number of P. intermedia species. (e) Total number of P. gulae species. (f) Total number of C. rectus species.

 

Journal of Dental Research, Vol. 86, No. 11, 1078-1082 (2007)
DOI: 10.1177/154405910708601111
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