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Journal of Dental Research
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Role of K+ATP Channels, Endothelin A Receptors, and Effect of Angiotensin II on Blood Flow in Oral Tissues

E. Berggreen* and K.J. Heyeraas

Department of Physiology, Årstadveien 19, University of Bergen, N-5009 Bergen, Norway;


Figure 1
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  		Figure 1. Original simultaneous recordings of systemic arterial pressure (PA), pulpal blood flow (PBF), and gingival blood flow (GBF), before, during, and after infusion of glibenclamid. Start of infusion of glibenclamid (40 µM, 0.3 mL, 1 min) indicated with an arrow. To observe the duration of the drop in PBF, we omitted electrical tooth stimulation in this experiment.

 

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  		Figure 2. Effects of glibenclamid infusions on pulpal blood flow (PBF), gingival blood flow (GBF), and mean arterial pressure (MAP) as % of control measurements (8 animals, 14 observations). Values are mean ± SEM. ***p < 0.001, **p < 0.01, *p < 0.05.

 

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  		Figure 3. Effects of electrical tooth stimulation on pulpal blood flow (PBF) and mean arterial pressure (MAP) before and after glibenclamid infusions. Values are mean ± SEM as % of control measurements (5 animals, 9 observations). No significant difference was observed between the measurements before and those after glibenclamid infusions.

 

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  		Figure 4. Original simultaneous recordings of phasic systemic arterial pressure (PA), pulpal blood flow (PBF), and gingival blood flow (GBF). Arrow points at start of infusion of angiotensin II.

 

Journal of Dental Research, Vol. 82, No. 1, 33-37 (2003)
DOI: 10.1177/154405910308200108
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